How does the body differentiate between hypovolemia and hyponatremia at the level of osmoreceptors and baroreceptors, and how does this affect ADH secretion?

We know that both hypovolemia (low blood volume) and hyponatremia (low sodium) can stimulate ADH release, but via different pathways baroreceptors and osmoreceptors respectively.

But what happens when both occur together, like in SIADH or heart failure? Which input dominates in regulating ADH? And how does this interaction influence fluid therapy decisions?

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